Do you really want to live forever? Futurist Ray Kurzweil has predicted that humans will achieve immortality in just seven years. Genetic engineering company touts ‘Jurassic Park’-like plan to ‘de-extinct’ dodo bird Elon Musk ‘comfortable’ putting Neuralink chip into one of his kids.
Read more ❯.
One can only hope.
A former Google engineer has just predicted that humans will achieve immortality in eight years, something more than likely considering that 86% of his 147 predictions have been correct.
Ray Kurzweil visited the YouTube channel Adagio, in a discussion on the expansion of genetics, nanotechnology and robotics, which he believes will lead to age-reversing ‘nanobots’.
A study finds that fruit from the cocklebur plant, an invasive weed, may have anti-aging and anti-inflammatory capabilities and could potentially be used in skin care.
Pensions behave as government mandated ponzi schemes. New contributors are needed to pay for past contributors. But what if there are less and less new contributors and contributions? And what if past generations live longer and longer lives?
Limited time: get 5 free stocks when you sign up to moomoo and deposit $100 and 15 free stocks when you deposit $1,000. Use link https://j.moomoo.com/00iPZo.
Continue reading “The Impending Collapse of the French Economy” »
University of Pittsburgh.
A metamaterial is any material engineered to have a property that is elusive to naturally occurring materials. The research introduces the use of metamaterials in the creation of concrete, providing the option to alter its brittleness, flexibility, and shapeability to allow builders to use less of the material without sacrificing strength or longevity.
The researchers add that these data demonstrate that CRISPRa is generally applicable across chromatin states and cell types, and highlights the factors that impact the degree of gene activation and how easy it is to reproduce the effects. Understanding these factors is important in the design and analysis of CRISPRa screens, which are used to look for genes involved in genetic diseases, the team points out.
Further study is required to continue to add to these rules and to see whether different CRISPRa or CRISPR interference techniques behave in a similar way.
“Our research has established a system for reporting the effectiveness of CRISPR activation in stem cells, allowing us to gain a better understanding of how CRISPRa works in multiple cell states,” says Qianxin Wu, PhD, first author from Wellcome Sanger. “We also showed that CRISPR gene activation is powerful enough to induce stem cells to differentiate into other cell states. This suggests that CRISPRa screens can be used to search for genes involved in cellular processes or to generate more accurate models of cell types in the body, aiding research into genetic diseases and regenerative medicine.”
Having both diabetes and tooth loss contributes to worse cognitive function and faster cognitive decline in older adults, according to a new study published in a special issue of the Journal of Dental Research focused on aging and oral health.
“Our findings underscore the importance of dental care and diabetes management for older adults in reducing the devastating personal and societal costs of Alzheimer’s disease and other related dementias,” said Bei Wu, vice dean for research at NYU Rory Meyers College of Nursing and co-director of the NYU Aging Incubator, as well as the study’s lead author.
Diabetes is a known risk factor for cognitive decline and dementia. Several of the hallmarks of diabetes —high blood sugar, insulin resistance, inflammation, and related heart disease—are thought to contribute to changes in the brain.
Michael Levin is a biologist at Tufts University working on novel ways to understand and control complex pattern formation in biological systems.
Michael Levin links.
Michael’s Twitter: https://twitter.com/drmichaellevin.
Michael’s Website: https://drmichaellevin.org.
Senescent cells accumulate with age when one senescent cell turns another cell senescent through SASP secretion. New research is offering new hope for fighting this.
Changes in the brain caused by Alzheimer’s disease are associated with shortening of the telomeres—the protective caps on the ends of chromosomes that shorten as cells age—according to a new study led by Anya Topiwala of Oxford Population Health, part of the University of Oxford, UK, published March 22 in the open-access journal PLOS ONE.
Telomeres on chromosomes protect DNA from degrading, but every time a cell divides, the telomeres lose some of their length. Short telomeres are a sign of stress and cellular aging, and are also associated with a higher risk of neurological and psychiatric disorders. Currently, little is known about the links between telomere length and changes that occur in the brains of people with neurological conditions. Understanding those relationships could offer insights into the biological mechanisms that cause neurodegenerative disorders.
In the new study, researchers compared telomere length in white blood cells to results from brain MRIs and electronic health records from more than 31,000 participants in the UK Biobank, a large-scale biomedical database and research resource containing anonymized genetic, lifestyle and health information from half a million UK participants.
